Eickelberg,Oliver

Institution: University of Giessen Lung Center, Germany

Research Interests: Lung, Fibrosis, Pulmonary Fibrosis, Cell, Therapeutic, Disease, Cells, Fibroblast, Idiopathic Pulmonary Fibrosis, Cancer, Lung Cancer, Extracellular Matrix, Epithelial Cell, Epithelial Cells, Gene, Growth, Human, Proteins, Transcription Factors, Transforming Growth Factor

Publications

Information and abstracts for author publications
Published Date	Impact	Publication
08/20/2012	38.3	Lancet New cellular and molecular mechanisms of lung injury and fibrosis in idiopathic pulmonary fibrosis.	Abstract
03/16/2009	13.1	The Journal of clinical investigation WNT1-inducible signaling protein-1 mediates pulmonary fibrosis in mice and is upregulated in humans with idiopathic pulmonary fibrosis.	Abstract
10/22/2009	6.8	Thorax SNAI transcription factors mediate epithelial-mesenchymal transition in lung fibrosis.	Abstract
07/29/2010	6.8	Thorax Increased expression of 5-hydroxytryptamine2A/B receptors in idiopathic pulmonary fibrosis: a rationale for therapeutic intervention.	Abstract
03/22/2012	5.9	The European respiratory journal What shall we do with the damaged proteins in lung disease? Ask the proteasome!	Abstract
03/27/2009	5.1	American journal of respiratory cell and molecular biology WNT signaling in lung disease: a failure or a regeneration signal?	Abstract
06/02/2014	5.1	American journal of respiratory cell and molecular biology Cigarette smoke-induced disruption of bronchial epithelial tight junctions is prevented by transforming growth factor-β.	Abstract
07/09/2012	4.8	The Journal of biological chemistry Zyxin is a transforming growth factor-β (TGF-β)/Smad3 target gene that regulates lung cancer cell motility via integrin α5β1.	Abstract
03/04/2009	4.5	Proteomics Subcellular fractionation of TGF-beta1-stimulated lung epithelial cells: a novel proteomic approach for identifying signaling intermediates.	Abstract
05/07/2013	4.1	PloS one Gli1 mediates lung cancer cell proliferation and Sonic Hedgehog-dependent mesenchymal cell activation.	Abstract